| ○種別 (必須): | □ | 学術論文 (審査論文)
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| ○言語 (必須): | □ | 英語
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| ○著者 (必須): | 1. | 山田 晃嗣 ([徳島大学.大学院社会産業理工学研究部.生物資源産業学域.生物生産系.生物資源生産科学分野])
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| 2. | (英) Saijo Yusuke (日) (読)
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| 3. | (英) Nakagami Hirofumi (日) (読)
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| 4. | (英) Takano Yoshitaka (日) (読)
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| ○題名 (必須): | □ | (英) Regulation of sugar transporter activity for antibacterial defense in Arabidopsis. (日)
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| ○要約 (任意): | □ | (英) Microbial pathogens strategically acquire metabolites from their hosts during infection. Here we show that the host can intervene to prevent such metabolite loss to pathogens. Phosphorylation-dependent regulation of sugar transport protein 13 (STP13) is required for antibacterial defense in the plant Arabidopsis thaliana STP13 physically associates with the flagellin receptor flagellin-sensitive 2 (FLS2) and its co-receptor BRASSINOSTEROID INSENSITIVE 1-associated receptor kinase 1 (BAK1). BAK1 phosphorylates STP13 at threonine 485, which enhances its monosaccharide uptake activity to compete with bacteria for extracellular sugars. Limiting the availability of extracellular sugar deprives bacteria of an energy source and restricts virulence factor delivery. Our results reveal that control of sugar uptake, managed by regulation of a host sugar transporter, is a defense strategy deployed against microbial infection. Competition for sugar thus shapes host-pathogen interactions. (日)
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| ○キーワード (推奨): |
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| ○誌名 (必須): | □ | Science (The American Association for the Advancement of Science)
(pISSN: 0036-8075, eISSN: 1095-9203)
| ○ISSN (任意): | □ | 1095-9203
ISSN: 0036-8075
(pISSN: 0036-8075, eISSN: 1095-9203) Title: Science (New York, N.Y.)Title(ISO): SciencePublisher: Amer. Assoc. Adv. Sci., Washington, DC. (NLM Catalog)
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| ○巻 (必須): | □ | 354
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| ○号 (必須): | □ | 6318
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| ○頁 (必須): | □ | 1427 1430
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| ○年月日 (必須): | □ | 西暦 2016年 12月 16日 (平成 28年 12月 16日)
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| ○URL (任意): |
| ○DOI (任意): | □ | 10.1126/science.aah5692 (→Scopusで検索)
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| ○PMID (任意): | □ | 27884939 (→Scopusで検索)
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| ○備考 (任意): | 1. | (英) PublicationType: Journal Article (日)
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