『徳島大学 教育・研究者情報データベース (EDB)』---[学外] /
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種別 (必須): 学術論文 (審査論文) [継承]
言語 (必須): 英語 [継承]
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著者 (必須): 1.油形 公則
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2.松井 好人
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3. (英) Shukunami C (日) (読)
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[継承]
4. (英) Takimoto A (日) (読)
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5.廣橋 紀 ([徳島大学.病院.中央診療施設等.リハビリテーション部]/->個人[佐藤 紀])
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6. (英) Ohtani O (日) (読)
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7. (英) Kimura T (日) (読)
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8. (英) Hiraki Y (日) (読)
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9.安井 夏生
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題名 (必須): (英) Differential expression of Tenomodulin and Chondromodulin-1 at the insertion site of the tendon reflects a phenotypic transition of the resident cells.  (日)    [継承]
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要約 (任意): (英) The insertion site of the tendon to the skeletal element is hypovascular and is one of the most common sites of dysfunction in the musculoskeletal system. However, the resident cells have been poorly defined due to a lack of a specific marker for tenocytes. We previously reported that Tenomodulin (Tnmd) and Chondromodulin-1 (Chm1) are homologous angiogenesis inhibitors and predominantly expressed in the avascular region of tendons and cartilage, respectively. In this study, we analyzed the expression of Tnmd, Chm1, alpha 1 chain of the type I collagen (Col1a1) and alpha 1 chain of the type II collagen (Col2a1) at the insertion site of the Achilles, patellar, or rotator cuff tendons of 1-week-old rabbits by in situ hybridization analysis. Tnmd was co-expressed with Col1a1 in tenocytes of these tendons, while Chm1 and Col2a1 were detected in chondrocytes of the hyaline cartilage. Interestingly, the cell population between Tnmd/Col1a1 positive tenocytes and Chm1/Col2a1 positive chondrocytes expressed Col1a1 but none of the other markers (Tnmd, Chm1, and Col2a1). Red blood cells were exclusively present at the interface between the tendon substance and cartilage in the insertion site of the Achilles tendon. Lack of Tnmd and Chm1 in this newly characterized cell population may allow the transitional zone between the poorly vascularized tendon and cartilage to establish the unique vascular pattern for blood supply.  (日)    [継承]
キーワード (推奨): 1. (英) Animals (日) (読) [継承]
2. (英) Animals, Newborn (日) (読) [継承]
3. (英) Cartilage (日) (読) [継承]
4. (英) Chondrocytes (日) (読) [継承]
5. (英) Cloning, Molecular (日) (読) [継承]
6. (英) Collagen Type I (日) (読) [継承]
7. (英) Collagen Type II (日) (読) [継承]
8. (英) Connective Tissue Cells (日) (読) [継承]
9. (英) Erythrocytes (日) (読) [継承]
10. (英) In Situ Hybridization (日) (読) [継承]
11. (英) Intercellular Signaling Peptides and Proteins (日) (読) [継承]
12. (英) Membrane Proteins (日) (読) [継承]
13. (英) Phenotype (日) (読) [継承]
14. (英) RNA, Messenger (日) (読) [継承]
15. (英) Rabbits (日) (読) [継承]
16. (英) Tendons (日) (読) [継承]
17. (英) Weight-Bearing (日) (読) [継承]
発行所 (推奨):
誌名 (必須): Tissue & Cell ([Elsevier])
(pISSN: 0040-8166, eISSN: 1532-3072)

ISSN (任意): 1532-3072
ISSN: 0040-8166 (pISSN: 0040-8166, eISSN: 1532-3072)
Title: Tissue & cell
Title(ISO): Tissue Cell
Publisher: Elsevier Ltd.
 (NLM Catalog  (Scopus  (CrossRef (Scopus information is found. [need login])
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(必須): 42 [継承]
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(必須): 116 120 [継承]
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年月日 (必須): 西暦 2010年 3月 23日 (平成 22年 3月 23日) [継承]
URL (任意):
DOI (任意): 10.1016/j.tice.2010.02.002    (→Scopusで検索) [継承]
PMID (任意): 20334886    (→Scopusで検索) [継承]
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WOS (任意): 000278299500006 [継承]
Scopus (任意): 2-s2.0-77952009722 [継承]
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備考 (任意): 1.(英) Article.Affiliation: Department of Orthopedics, Institute of Health Biosciences, The University of Tokushima Graduate School, 3-18-15 Kuramoto, Tokushima 770-8503, Japan.  (日)    [継承]
2.(英) Article.PublicationTypeList.PublicationType: Journal Article  (日)    [継承]
3.(英) Article.PublicationTypeList.PublicationType: Research Support, Non-U.S. Gov't  (日)    [継承]

標準的な表示

和文冊子 ● Kiminori Yukata, Yoshito Matsui, C Shukunami, A Takimoto, Nori Hirohashi, O Ohtani, T Kimura, Y Hiraki and Natsuo Yasui : Differential expression of Tenomodulin and Chondromodulin-1 at the insertion site of the tendon reflects a phenotypic transition of the resident cells., Tissue & Cell, Vol.42, No.2, 116-120, 2010.
欧文冊子 ● Kiminori Yukata, Yoshito Matsui, C Shukunami, A Takimoto, Nori Hirohashi, O Ohtani, T Kimura, Y Hiraki and Natsuo Yasui : Differential expression of Tenomodulin and Chondromodulin-1 at the insertion site of the tendon reflects a phenotypic transition of the resident cells., Tissue & Cell, Vol.42, No.2, 116-120, 2010.

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