『徳島大学 教育・研究者情報データベース (EDB)』---[学外] /
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種別 (必須): 学術論文 (審査論文) [継承]
言語 (必須): 英語 [継承]
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審査 (推奨): Peer Review [継承]
カテゴリ (推奨):
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組織 (推奨): 1.徳島大学.工学部.生物工学科.生物反応工学講座 [継承]
著者 (必須): 1. (英) Wakana Yuichi (日) (読)
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2. (英) Koyama Satoshi (日) (読)
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3. (英) Nakajima Ken-ichi (日) (読)
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4. (英) Hatsuzawa Kiyotaka (日) (読)
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5.長浜 正巳
役割 (任意): (英)   (日) 考察   [継承]
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6. (英) Tani Katsuko (日) (読)
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7. (英) Hauri Hans-Peter (日) (読)
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8. (英) Melancon Paul (日) (読)
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9. (英) Tagaya Mitsuo (日) (読)
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題名 (必須): (英) Reticulon 3 is involved in membrane trafficking between the endoplasmic reticulum and Golgi  (日)    [継承]
副題 (任意):
要約 (任意): (英) Reticulons (RTNs) constitute a family of endoplasmic reticulum (ER)-associated proteins with a reticular distribution. Despite the implication of their neuronal isoforms in axonal regeneration, the function of their widely expressed isoforms is largely unknown. In this study, we examined the role of the ubiquitously expressed RTN3 in membrane trafficking. Ectopically expressed RTN3 exhibited heterogeneous patterns; filamentous, reticular, and granular distributions. The ER morphology changed accordingly. In cells where RTN3 displayed a filamentous/reticular distribution, protein transport between the ER and Golgi was blocked, and Golgi proteins were dispersed. In contrast, ERGIC-53, a marker for the ER-Golgi intermediate compartment, accumulated at the perinuclear region, and remained there even after cells were treated with agents that induce redistribution of Golgi proteins to the ER, indicating an inhibition of Golgi-to-ER transport of ERGIC-53. These results suggest that RTN3 plays a role in membrane trafficking in the early secretory pathway.  (日)    [継承]
キーワード (推奨): 1. (英) Bafilomycin A1 (日) (読) [継承]
2. (英) Brefeldin A (日) (読) [継承]
3. (英) Endoplasmic reticulum (日) (読) [継承]
4. (英) ERIGC-53 (日) (読) [継承]
5. (英) Golgi apparatus (日) (読) [継承]
6. (英) Nocodazole (日) (読) [継承]
7. (英) Reticulon (日) (読) [継承]
8. (英) Vesicular transport (日) (読) [継承]
発行所 (推奨): Elsevier (->組織[Elsevier Science]) [継承]
誌名 (必須): Biochemical and Biophysical Research Communications ([Elsevier])
(pISSN: 0006-291X, eISSN: 1090-2104)

ISSN (任意): 0006-291X
ISSN: 0006-291X (pISSN: 0006-291X, eISSN: 1090-2104)
Title: Biochemical and biophysical research communications
Title(ISO): Biochem Biophys Res Commun
Publisher: Elsevier Inc.
 (NLM Catalog  (Scopus  (CrossRef (Scopus information is found. [need login])
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(必須): 334 [継承]
(必須): 4 [継承]
(必須): 1198 1205 [継承]
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年月日 (必須): 西暦 2005年 9月 9日 (平成 17年 9月 9日) [継承]
URL (任意): http://dx.doi.org/10.1016/j.bbrc.2005.07.012 [継承]
DOI (任意): 10.1016/j.bbrc.2005.07.012    (→Scopusで検索) [継承]
PMID (任意): 16054885    (→Scopusで検索) [継承]
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備考 (任意): 1.(英) Article.Affiliation: School of Life Science, Tokyo University of Pharmacy and Life Science, Hachioji, Tokyo 192-0392, Japan.  (日)    [継承]
2.(英) Article.PublicationTypeList.PublicationType: Journal Article  (日)    [継承]
3.(英) Article.PublicationTypeList.PublicationType: Research Support, Non-U.S. Gov't  (日)    [継承]

標準的な表示

和文冊子 ● Yuichi Wakana, Satoshi Koyama, Ken-ichi Nakajima, Kiyotaka Hatsuzawa, Masami Nagahama, Katsuko Tani, Hans-Peter Hauri, Paul Melancon and Mitsuo Tagaya : Reticulon 3 is involved in membrane trafficking between the endoplasmic reticulum and Golgi, Biochemical and Biophysical Research Communications, Vol.334, No.4, 1198-1205, 2005.
欧文冊子 ● Yuichi Wakana, Satoshi Koyama, Ken-ichi Nakajima, Kiyotaka Hatsuzawa, Masami Nagahama, Katsuko Tani, Hans-Peter Hauri, Paul Melancon and Mitsuo Tagaya : Reticulon 3 is involved in membrane trafficking between the endoplasmic reticulum and Golgi, Biochemical and Biophysical Research Communications, Vol.334, No.4, 1198-1205, 2005.

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