『徳島大学 教育・研究者情報データベース (EDB)』---[学外] /
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EID=130831EID:130831, Map:0, LastModified:2018年3月20日(火) 16:01:39, Operator:[三木 ちひろ], Avail:TRUE, Censor:0, Owner:[吉田 守美子], Read:継承, Write:継承, Delete:継承.
種別 (必須): 学術論文 (審査論文) [継承]
言語 (必須): 英語 [継承]
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組織 (推奨):
著者 (必須): 1. (英) Inoue Nami (日) (読)
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[継承]
2. (英) Kunishige Makoto (日) (読)
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3.吉田 守美子 ([徳島大学.大学院医歯薬学研究部.医学域.医科学部門.内科系.血液・内分泌代謝内科学])
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4. (英) Oshima Yasushi (日) (読)
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5. (英) Ohnishi Yoshiaki (日) (読)
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6. (英) Kuroda Yasuhiro (日) (読)
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7. (英) Asano Atsuko (日) (読)
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8. (英) Yoshino Hiide (日) (読)
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9.松本 俊夫
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10.三ツ井 貴夫
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題名 (必須): (英) Dissociation between titer of anti-ganglioside antibody and severity of symptoms in a case of Guillain-Barre syndrome with treatment-related fluctuation  (日)    [継承]
副題 (任意):
要約 (任意): (英) Since plasma exchange (PE) and intravenous immunoglobulin (i.v.Ig) have been widely used in treatment for Guillain-Barré syndrome (GBS), early relapse and treatment-related fluctuation have been a potential problem, but little is known about the mechanism of relapse and fluctuation. We describe a patient who had GBS with treatment-related fluctuation. A 37-year-old Japanese man exhibited acute distal-dominant weakness in upper limbs after upper respiratory infection. His cranial nerve system was normal and muscle weakness was limited to upper limbs. Anti-GT1a IgG was strongly positive and anti-GQ1b IgG was also detected in his serum. Muscle weakness responded well to double-filtration plasmapheresis (DFPP) followed by i.v.Ig, but relapsed 45 days after the initial treatment. Although repeated treatments were effective, the patient showed additional minor deterioration twice. Motor nerve conduction velocities (MCVs) corresponded to the muscle weakness, but elevated level of cerebrospinal fluid (CSF) protein remained and anti-ganglioside antibody titers steadily decreased throughout the clinical course. These findings indicate that the clinical fluctuation was not due to changes in the production of anti-ganglioside antibodies but presumably to the transient beneficial effects of DFPP/i.v.Ig and the outlasting inflammatory response in peripheral nerves.  (日)    [継承]
キーワード (推奨): 1. (英) Anti-GT1a antibody (日) (読) [継承]
2. (英) anti-GQ1b antibody (日) (読) [継承]
3. (英) acute inflammatory demyelinating (日) (読) [継承]
4. (英) Intravenous immunoglobulin (日) (読) [継承]
5. (英) Plasma exchange (日) (読) [継承]
発行所 (推奨): Elsevier (->組織[Elsevier Science]) [継承]
誌名 (必須): Journal of the Neurological Sciences (World Federation of Neurology)
(pISSN: 0022-510X, eISSN: 1878-5883)

ISSN (任意): 0022-510X
ISSN: 0022-510X (pISSN: 0022-510X, eISSN: 1878-5883)
Title: Journal of the neurological sciences
Title(ISO): J Neurol Sci
Publisher: Elsevier BV
 (NLM Catalog  (Scopus  (CrossRef (Scopus information is found. [need login])
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(必須): 210 [継承]
(必須): 1-2 [継承]
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年月日 (必須): 西暦 2003年 6月 15日 (平成 15年 6月 15日) [継承]
URL (任意): http://dx.doi.org/10.1016/S0022-510X(03)00031-5 [継承]
DOI (任意): 10.1016/S0022-510X(03)00031-5    (→Scopusで検索) [継承]
PMID (任意): 12736098    (→Scopusで検索) [継承]
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WOS (任意): 000183107300021 [継承]
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備考 (任意): 1.(英) Article.Affiliation: First Department of Internal Medicine, University of Tokushima School of Medicine, 3 Kuramoto-cho, Tokushima, Tokushima 770-8503, Japan.  (日)    [継承]
2.(英) Article.PublicationTypeList.PublicationType: Case Reports  (日)    [継承]
3.(英) Article.PublicationTypeList.PublicationType: Journal Article  (日)    [継承]

標準的な表示

和文冊子 ● Nami Inoue, Makoto Kunishige, Sumiko Yoshida, Yasushi Oshima, Yoshiaki Ohnishi, Yasuhiro Kuroda, Atsuko Asano, Hiide Yoshino, Toshio Matsumoto and Takao Mitsui : Dissociation between titer of anti-ganglioside antibody and severity of symptoms in a case of Guillain-Barre syndrome with treatment-related fluctuation, Journal of the Neurological Sciences, Vol.210, No.1-2, 105-108, 2003.
欧文冊子 ● Nami Inoue, Makoto Kunishige, Sumiko Yoshida, Yasushi Oshima, Yoshiaki Ohnishi, Yasuhiro Kuroda, Atsuko Asano, Hiide Yoshino, Toshio Matsumoto and Takao Mitsui : Dissociation between titer of anti-ganglioside antibody and severity of symptoms in a case of Guillain-Barre syndrome with treatment-related fluctuation, Journal of the Neurological Sciences, Vol.210, No.1-2, 105-108, 2003.

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